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The 46th Annual Meeting of The Japanese Society for ImmunologyProgram for PosterSession TitleDateTimeSubmission IDProgram AuthorAffiliationAbstract Title1002981-A-W1-1-O/PSho SHIBATAD epartment of Immune Regulation, Tokyo Medical and Dental University, Tokyo, JapanMonocyte-derived macrophages in the lung contribute to the development of pulmonary emphysemathrough MMP-12 production in a mouse model of COPD1003011-A-W1-2-PMinoru TakeuchiDept. of Animal Medical Science, Kyoto Sangyo University, Kyoto, Japan<strong>Effect of H</strong> <strong>ot Water Extract from Agaricus Blazei Murill onLipopolysaccharide (LPS) -induced Lung Inflammation and the Immunological Mechanism</strong>1000411-A-W1-3-O/PSatoko Tahara-HanaokaDepartment of Immunology, Faculty of Medicine, University of Tsukuba, Tsukuba,JapanInhibition of house dust mite-induced Th2 responses by Allergin-1 immunoreceptor on dendritic cells1001001-A-W1-4-O/PKoji HayashizakiDepartment of Immunology, Graduate School of Medicine, Chiba University, Chiba,JapanMyosin light chains 9 and 12 are functional ligands for CD69 that regulate airway inflammation1002351-A-W1-5-PRyuichi NagashimaDivision of Cancer Biology and Therapeutics, Miyagi Cancer Certer Reserch Institute,Natori, Japan<strong>Nrf2 activation suppressed lung allergic inflammation induced by

The 46th Annual Meeting of The Japanese Society for Immunology Program for Poster Session Title Date Time Submission ID Program No. 1st Author Affiliation Abstract Title 100040 1-B-W4-1-O/P Ayako Wakabayashi Department of Microbiology and Immunology, Nippon Medical School, Tokyo, Japan Enhancement of epithelial cell death, cytoplasmic HMGB1 expression in IECs, and increase of HMGB1

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Transcription of Session Title Date Time Submission ID Program …

1 The 46th Annual Meeting of The Japanese Society for ImmunologyProgram for PosterSession TitleDateTimeSubmission IDProgram AuthorAffiliationAbstract Title1002981-A-W1-1-O/PSho SHIBATAD epartment of Immune Regulation, Tokyo Medical and Dental University, Tokyo, JapanMonocyte-derived macrophages in the lung contribute to the development of pulmonary emphysemathrough MMP-12 production in a mouse model of COPD1003011-A-W1-2-PMinoru TakeuchiDept. of Animal Medical Science, Kyoto Sangyo University, Kyoto, Japan<strong>Effect of H</strong> <strong>ot Water Extract from Agaricus Blazei Murill onLipopolysaccharide (LPS) -induced Lung Inflammation and the Immunological Mechanism</strong>1000411-A-W1-3-O/PSatoko Tahara-HanaokaDepartment of Immunology, Faculty of Medicine, University of Tsukuba, Tsukuba,JapanInhibition of house dust mite-induced Th2 responses by Allergin-1 immunoreceptor on dendritic cells1001001-A-W1-4-O/PKoji HayashizakiDepartment of Immunology, Graduate School of Medicine, Chiba University, Chiba,JapanMyosin light chains 9 and 12 are functional ligands for CD69 that regulate airway inflammation1002351-A-W1-5-PRyuichi NagashimaDivision of Cancer Biology and Therapeutics, Miyagi Cancer Certer Reserch Institute,Natori, Japan<strong>Nrf2 activation suppressed lung allergic inflammation induced by type2-innate lymphoidcells.

2 </strong>1004411-A-W1-6-PJung Yeon HongDepartment of Pediatrics and Institute of Allergy, Brain Korea 21 PLUS Project forMedical Science, Yonsei University College of Medicine, Seoul, Korea<strong>Chitotriosidase Regulation of Allergen-induced Airway Inflammation AirwaysHyperresponsiveness and Foxp3<sup>+</sup> Treg Cells</strong>1004501-A-W1-7-PMi Na KimDepartment of Pediatrics and Institute of Allergy, Brain Korea 21 PLUS Project forMedical science, Yonsei University College of Medicine, Seoul, Republic of KoreaALCAM on dendritic cell contributes Th2 response in allergic airway inflammation1003211-A-W1-8-PTomohiro TamachiDepartment of Allergy and Clinical Immunology, Graduate School of Medicine, ChibaUniversity, Chiba, JapanA20 (<em>Tnfaip3</em>) expressed in T cells suppresses Th2 cell-mediated allergic airwayinflammation in mice1001891-A-W1-9-O/PAiko SakuDepartment of Allergy and Clinical Immunology, Graduate School of Medicine, ChibaUniversity, Chiba, JapanLung epithelial fucosylation promotes the development of house dust mite (HDM)-induced allergicairway MiyasakaDivision of Pathophysiology, Department of Pharmaceutical Sciences, Faculty ofPharmaceutical Sciences, Tohoku Medical and Pharmaceutical University, Sendai,JapanPossible involvement of CD103<sup>+</sup> dendritic cells in female-predominant Th2 priming inasthma1003561-A-W1-11-O/PKentaro FujiwaraDept.

3 Of Immunology<strong>M1 macrophages mediate attenuation of bronchial asthma by matrix-metalloproteinase-2</strong>1004731-A-W1-12-PAsako TanoueDepartment of Pulmonary Medicine, Graduate School of Medical and Dental Sciences,Kagoshima University, Kagoshima, JapanThe role of group III secreted phospholipase A2 in allergic asthma1000311-A-W1-13-PChika KanamoriDepartment of Animal medical science, Faculty of Life science, Kyoto SangyoUniversity, Kyoto, Japan<strong>Effects of Cryptomeria Japonica pollen and cigarette smoke on immune cells</strong>1001991-A-W1-14-O/PKatsuhide OkunishiInstitute for Molecular and Cellular Regulation, Gunma University, Maebashi, JapanRab27 effector exophilin5 regulates IL-33 signaling in allergic immune responses1005901-A-W1-15-PMasashi IkutaniDepartment of Immune Regulation, Research Institute, National Center for GlobalHealth and Medicine, Chiba, JapanChronic IL-33-induced inflammation results in pulmonary arterial hypertrophy1002181-A-W1-16-PPrince Baffour TontoDept.

4 Of Immunology<strong>Role of eosinophils and metalloproteinase-2 in airway remodeling</strong>1002911-A-W1-17-O/PKazuyuki NakagomeDepartment of Respiratory Medicine and Allergy Center, Saitama Medical University,Saitama, JapanElevated periostin concentrations in the bronchoalveolar lavage fluid of eosinophilic pneumonia1003391-A-W1-18-PShigeki KatohDepartment of Respiratory Medicine, Kawasaki Medical School, Okayama, JapanPossible role of IL-25 in eosinophilic lung inflammation in patients with eosinophilic pneumonia1000561-A-W2-1-POji NakamuraDepartment of Applied Biological Chemistry, Tokyo University of Agriculture andTechnology, Tokyo, JapanEffect of honeybush extracts on the murine model of pollen allergy1000641-A-W2-2-PInoue AkikoDepartment of Otorhinolaryngology, Toho University School of Medicine, Tokyo, JapanStudy of relationship of allergy and chronic rhinosinusitis1001531-A-W2-3-PDong-Young KimDepartment of Otorhinolaryngology-Head and Neck Surgery, Seoul National UniversityCollege of Medicine, Seoul, KoreaAnti-allergic effect of intranasal vitamin D treatment by inhibiting dendritic cell activation in the allergicrhinitis mouse model1002491-A-W2-4-PAyumi FukuokaDepartment of Immunology, Hyogo College of Medicine, Nishinomiya, Hyogo, JapanCystatin SN inhibits Japanese cedar pollen-induced allergic rhinitis in MatsuiDepartment of Fundamental Biosciences, Shiga University of Medical Science, Otsu,JapanRole of neutrophils in the nasal mucosa in a mouse model of allergic rhinitis1002921-A-W2-6-PDaisuke NagakuboDepartment of Fundamental Biosciences, Shiga University of Medical Science, Otsu.

5 JapanInvolvement of CCL28 in the pathogenesis of allergic rhinitis in a mouse model1003001-A-W2-7-O/PMasaya MatsudaLaboratory of Immunopharmacology, Faculty of Pharmaceutical Sciences, SetsunanUniversity, Osaka, JapanEffects of subcutaneous immunotherapy (SCIT) on regulatory T cells in pollinosis patients1004841-A-W2-8-O/PKazuya TakedaLaboratory of Immune regulation, Immunology Frontier Research center, OsakaUniversity, Suita, JapanEctopic T-B interaction for antigen-specific IgE antibody responses in nasal polyps of Eosinophilicchronic rhinosinusitis patients1001101-A-W2-9-O/PDat Nguyen-TienDivision of Molecular Biology, Research Institute for Biomedical Sciences, TokyoUniversity of Science<strong>A selection technique for human IgE<sup>+</sup> memory B cells and the function of the new isoform of human IgE</strong>1001131-A-W2-10-O/PMariana AlmeidaDepartment of Immunology, Faculty of Medicine, University of Tsukuba, Tsukuba,JapanInhibition of house dust mite-induced MAP kinase activation in dendritic cells by Allergin-1immunoreceptor1002031-A-W2-11- PHirotomo ShibaguchiDepartment of Biochemistry, Faculty of Medicine.

6 Fukuoka University, Fukuoka, JapanGene analysis of HL-60 cells and neutrophils to generate ideal chemotactic cells for HiSAT1002051-A-W2-12-O/PShihono KatoLaboratory for Innate Immune Systems, RIKEN, IMS, Kanagawa, JapanRole of ILC2 in the formation of maternal allergic inflammation and allergic susceptibility of the offspring1002321-A-W2-13-O/PMio OhashiDepartment of Immunoregulation, Institute of Medical Science, Tokyo MedicalUniversity, Tokyo, JapanPrediction of Chemical Respiratory and Contact Sensitizers by OX40L Expressionin Dendritic CellsUsing a Novel 3D Co-Culture System1003111-A-W2-14-O/PKano TanabeDepartment of Medical Technology, Kumamoto Health Science University, Kumamoto,JapanCaMKII regulates IgE class switch recombination via TRAF3 ubiquitination1003171-A-W2-15-PAkira AwayaDermatology&Epidemiology Research Institute(DERI),Totsuka,Yokohama, JapanStrategy for advance of melanocyte immunology from research on pollen-induced diseases(PID) suchas Kawasaki disease(KD) - Epidemiological study of KD(13): Statistical analyses reveal epidemics ofInfluenza suppress or postpone KD onsets.

7 Coadministration1005191-A-W2-16-O/PAyako TakamoriAtopy (Allergy) Research Center, Juntendo University School of Medicine, Tokyo,Japan<strong>Ceramide-CD300f interaction inhibits Mrgprb2-mediated mast cell activation and pseudo-allergic drug reactions in mice</strong>1005531-A-W2-17-PNarumi SuzukiLaboratory for Cytokine Regulation,Research Center for Integrative Medical Science,RIKEN Yokohama Institute,Yokohama,japanE4BP4/NFIL-3 is a critical transcription factor to control TH2 attraction into inflammation site duringchronic allergic SoichiroDepartment of Immune Regulation, Tokyo Medical and Dental University, Tokyo, Japan<strong>Prolonged Ca<sup>2+</sup> influx via STIM2 is essential for cytokine-induced IL-4 productionin basophil</strong>1006141-B-W3-1-O/PRyota SatoDivision of Innate Immunity, The Institute of Medical Science, The University of Tokyo,Japan<strong>Requirement of glycosylation machinery in Toll-like receptor responses revealed byCRISPR/Cas9 screening </strong>1000701-B-W3-2-O/PSachiko Akashi-TakamuraAichi Medical University School of Medicine, Nagakute, JapanC4b binding protein negatively regulates cell-surface TLRs response1002161-B-W3-3-O/PKen TakashimaDepartment of Immunology, Graduate School of Medicine, Hokkaido University,Sapporo, JapanNovel Negative Regulators for MDA5-Mediated Innate Immune Response1006411-B-W3-4-O/PMichihiro TakahamaInstitute of Advanced Medical Sciences, Tokushima University, Tokushima, JapanRAB2B-GARIL5 complex promotes innate immune response against DNA virus1001881-B-W3-5-O/PNaoya SakaguchiResearch Institute for Microbial Diseases (RIMD)

8 , Osaka University, Osaka, Japan<strong>Role of GABARAP subfamily in the regulation of non-canonical inflammasomeactivation</strong>1003221-B-W3-6-O/PKohsuke TsuchiyaCancer Research Institute, Kanazawa University, Kanazawa, JapanCaspase-1 serves as an apoptosis-initiating caspase in the absence of Gasdermin D (GSDMD)1001811-B-W3-7-O/PHo Minh VanDepartment of Emergency and Critical Care Medicine, Graduate School of Biomedicaland Health Sciences, Hiroshima University<strong>Detection of ATP-dependent inflammatory suppression of the innate immune response inseptic patients</strong>1000781-B-W3-8-O/PMasahiro NagataDepartment of Molecular Immunology, Research Institute for Microbial Diseases,Osaka University, Osaka, JapanIntracellular metabolite, -glucosylceramide, is an endogenous adjuvant acting through Mincle1006321-B-W3-9-PYuki NakajimaDepartment of Immunology, Graduate School of Medicine, The University of TokyoIdentificdation of immunogenic endogenous RNA and its inhibitor1006291-B-W3-10-PAsuka InoueResearch and Development, Sanofi , Tokyo.

9 JapanImmune regulation by inhibitory oligonucleotide ISM ODN for hmgb1 in inflammatory disease models1005221-B-W3-11-PMotoya MuraseGraduate School of Biological Sciences, Nara Institute of Science and Technology(NAIST), Nara, JapanIntravesicular acidification regulates lipopolysaccharide inflammation and tolerance through Toll-likereceptor 4 trafficking1003791-B-W3-12-PRina SugimotoDepartment of Cell Biological Science, Graduate School of Life Science, HokkaidoUniversity, Sapporo, JapanEvaluation system for receptor expression and function in the intestinal stem cell niche1005331-B-W3-13-PHideki SanjoDepartment of Molecular and Cellular Immunology, Shinshu University Schhol ofMedicine, Nagano, JapanLoss of TAK1 leads to TLR-driven macrophage cell death and inflammation that occur by a TNF-independent mechanism1000951-B-W3-14-PShimpei WatanabeR&D Innovation Group, Kracie Holdings, Ltd.

10 , Tokyo, JapanArctigenin suppresses the activation of NLRP3 and AIM2 AdachiDepartment of Immunology, Hyogo College of Medicine, Nishinomiya, Hyogo, JapanPurification and characterization of a unique DAMP of a potent inducer of IL-33 expression in non-hematopoietic cellsOdd Number /16:30-17:30 Even Number /17:30-18:30 Odd Number /16:30-17:30 Even Number /17:30-18:30 Odd Number /16:30-17:30 Even Number /17:30-18:30W3 Endogenous innateimmunity andinflammation-1 Dec. 12(Tue.), 2017W1 Allergy-1 : Lower airwayallergic inflammationDec. 12(Tue.), 2017W2 Allergy-2: Upper airwayallergic inflammation andallergic responsesDec. 12(Tue.), 20171 / 13 The 46th Annual Meeting of The Japanese Society for ImmunologyProgram for PosterSession TitleDateTimeSubmission IDProgram AuthorAffiliationAbstract Title1000401-B-W4-1-O/PAyako WakabayashiDepartment of Microbiology and Immunology, Nippon Medical School, Tokyo, JapanEnhancement of epithelial cell death, cytoplasmic hmgb1 expression in IECs, and increase of HMGB1release by oral CT-stimulation1005291-B-W4-2-O/PEri KumakiDepartment of Pediatrics and Developmental biology, Tokyo Medical and DentalUniversity Graduate School of Medical and Dental Sciences, Tokyo, JapanAbberant Neutrophil Function in <em>PSTPIP1</em> mutations1003251-B-W4-3-O/PAkiko IshitaniNara Medical University, Kashihara, Nara, Japan<em>The three faces of F.


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